Abstract: the size of the ventricular septal defect in children is very different, the smallest defect of the membrane is only 2 ~ 3 mm, the
Abstract: the size of the ventricular septal defect in children is very different, the smallest defect of the membrane is only 2 ~ 3 mm, the maximum is even more than and 20 mm. The harm and the lack of the size of the defect of congenital ventricular defect more closely, the greater the harm.
The difference of congenital ventricular missing, the minimum membrane defect of only 2 ~ 3 mm, more than and 20 mm or even the largest. The harm and the lack of the size of the defect of congenital ventricular defect more closely, the greater the harm.
If the lack of room is very small, there can be a part of the possibility of spontaneous closure of VSD, in muscular ventricular septal defect in three especially, tricuspid septal leaflet of the will and the surrounding tissue adhesion, the flow is to reduce the possibility of. Even without self closing, it usually does not cause serious health problems.
If the lack of room, in the months after birth can have a serious impact on children. At the beginning of the first 1-2 weeks after birth, with the right ventricular pressure drop, the blood flow began to flow from the left ventricle to the right ventricle, and gradually developed congestive heart failure. Pulmonary artery blood flow will increase continuously, the pulmonary vascular wall will therefore thickening, produce serious pulmonary hypertension consequences, later will develop as cyanosis. The pulmonary artery pressure increased in early stage was reversible, but gradually progressed to irreversible lesions.
The left ventricle is the blood pump of the aorta, and the pressure in the left ventricle of the left ventricle can reach 100mmHg (children) or higher (adult). Under normal circumstances, right ventricular systolic pressure in only 10 ~ 20mmHg, if there exists a ventricular septal defect, left ventricular blood will flow through the defect into the right ventricle, the flow rate depends on the size of the defect and the pressure difference between the two ventricle. The greater the pressure difference, the more the left ventricular blood flow to the right ventricle. Because the right ventricle is the blood pump of the pulmonary artery, the blood flow into the pulmonary artery increases after the right ventricle increases, causing the pulmonary artery to be congested.
The initial stage of pulmonary hypertension is dynamic, such as during this period for infants with congenital heart disease in children with heart surgery repair room missing, postoperative pulmonary artery pressure tends to drop to normal or normal. If there is a large ventricular septal defect or atrial septal defect and patent ductus arteriosus, the disease will develop rapidly. Because of the severe pulmonary hypertension, the pulmonary artery wall is thickened and hardened. At the end of the day, when the pulmonary artery pressure is higher than the aortic pressure, there is a right to left shunt. Left ventricular blood red blood oxygenation after, when left to right shunt when children with cyanotic lips cyanosis performance etc.. Once because of severe pulmonary hypertension and right to left shunt, right ventricle without oxygenated blood shunt to left ventricle, children will appear cyanosis. At this time, the child will lose the opportunity to operate, this time the surgery will only result in adverse consequences. Therefore, the best time to recommend surgery for congenital heart disease in children is the sooner the better!
Key words: Children's lack of ventricular damage in children with ventricular deficiency
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